Latanoprost is the first prostaglandin agent approved in the treatment of glaucoma and it is increasingly chosen as first-line therapy in all developed countries. of some concern is the ability of this agent to cause permanent iris color changes, conjunctival hyperemia and lash pigmentation.

Glaucoma is a group of eye disorders traditionally characterized by progressive damage to the eye, at least partly due to elevated intraocular pressure (IOP). Glaucoma affects 1 in 200 people aged 50 or younger and 1 in 10 over the age of 80. It is the leading cause of irreversible blindness in the world and the second leading cause of vision loss after cataract, which is reversible surgically. Primary open-angle glaucoma (POAG) is the most common form of glaucoma, accounting for about 60 to 70% of all glaucoma.

Latanoprost is being used alone or in combination with other agents for the treatment of glaucoma. The rationale for the development of a topical ocular product that combines a prostaglandin analogue (Latanoprost) and a s-blocker (Timolol) in a single formulation was to provide a better product with IOP-lowering efficacy that is greater than either component product alone, IOP-lowering efficacy that is similar to the concomitant administration of the component products, and convenience of once-daily dosing, thereby promoting better compliance.

Applications

To treat glaucoma and ocular hypertension

Pharmacology

Latanoprost is a prostanoid selective FP receptor agonist that is believed to reduce the intraocular pressure (IOP) by increasing the outflow of aqueous humor. Studies in animals and man suggest that the main mechanism of action is increased uveoscleral outflow. Elevated IOP represents a major risk factor for glaucomatous field loss. The higher the level of IOP, the greater the likelihood of optic nerve damage and visual field loss.

The production of Latanoprost is in compliance with ICH GMP; the GMP certificate has been obtained from Taiwan authority.

Sales to countries without patent issues only

Proprietary Names

XalatanR; XalcomR

References

1. Ishida N, Odani-Kawabata N, Shimazaki A,; Prostanoids in the therapy of glaucoma; Cardiovasc Drug Rev. 2006 Spring;24(1):1-10. Review.

2. Detry-Morel M.; Side effects of glaucoma medications; Bull Soc Belge Ophtalmol. 2006;(299):27-40. Review.

3. Holmstrom S, Buchholz P, Walt J, Wickstrom J, Aagren M.; Analytic review of bimatoprost, latanoprost and travoprost in primary open angle glaucoma; Curr Med Res Opin. 2005 Nov;21(11):1875-83. Review.

4. Grierson I, Jonsson M, Cracknell K.; Latanoprost and pigmentation; Jpn J Ophthalmol. 2004 Nov-Dec;48(6):602- 12. Review.

5. Feldman RM.; An evaluation of the fixed-combination of latanoprost and timolol for use in open-angle glaucoma and ocular hypertension; Expert Opin Pharmacother. 2004 Apr;5(4):909-21. Review.

6. Fechtner RD, Realini T.; Fixed combinations of topical glaucoma medications; Curr Opin Ophthalmol. 2004 Apr;15(2):132-5. Review.

7. Costagliola C, Parmeggiani F, Sebastiani A.; Assessing the cost-effectiveness of switching from a beta-blocker to latanoprost in the treatment of ocular hypertension; Expert Opin Pharmacother. 2003 Oct;4(10):1775-88. Review.

8. Brubaker RF.; Targeting outflow facility in glaucoma management; Surv Ophthalmol. 2003 Apr;48 Suppl 1:S17- 20. Review.

9. Perry CM, McGavin JK, Culy CR, Ibbotson T.; Latanoprost : an update of its use in glaucoma and ocular hypertension; Drugs Aging. 2003;20(8):597-630. Review.

10. Johnstone MA, Albert DM.; Prostaglandin-induced hair growth; Surv Ophthalmol. 2002 Aug;47 Suppl 1:S185-202. Review.

11. Grierson I, Pfeiffer N, Cracknell KP, Appleton P.; Histology and fine structure of the iris and outflow system following latanoprost therapy; Surv Ophthalmol. 2002 Aug;47 Suppl 1:S176-84. Review

 

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